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Therefore, it remains unclear whether the reduction of steatosis observed in animal and human trials in response to treatment with GLP-1R agonists results from direct activation of hepatic GLP-1R or the indirect impact such as weight loss, increased insulin sensitivity, brain-liver signals such as brain leptin 51 , or other hormonal signals that these agonists might trigger 14

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Its key roles include: Stimulating insulin secretion when blood glucose rises Reducing glucagon release (which helps limit excess glucose production) Slowing gastric emptying to promote satiety Supporting appetite regulation and weight balance Healthy GLP-1 signaling is associated with improved glycemic control, reduced cravings, and better metabolic flexibility

Research suggests GHK-CU has a relatively short half-life, making twice-daily administration theoretically superior for sustained gene expression effects